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Gymnema, leaves

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Gymnema, leaves

Additional Information

Botanical Name Gymnema sylvestris
Size 1 oz. (larger sizes available)

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Availability: In stock.

$ 13.35
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Quick Overview

Uses:
Reduces appetite for sweet food. Reduces absorption of sugars by the intestines. The leaves interfere with the ability to taste sweetness, which explains the Hindi name gurmar - "destroyer of sugar." Gymnema sylvestre is a stomachic, diuretic, refrigerant, astringent, and tonic. It has been found to increase urine output and reduce hyperglycemia in both animal and human studies.

Product Description

Body Parts Affected:
Tongue and Intestines.

Uses:
Reduces appetite for sweet food. Reduces absorption of sugars by the intestines. The leaves interfere with the ability to taste sweetness, which explains the Hindi name gurmar - "destroyer of sugar." Gymnema sylvestre is a stomachic, diuretic, refrigerant, astringent, and tonic. It has been found to increase urine output and reduce hyperglycemia in both animal and human studies.

Dose:
Liquid: Shake well. Take 20-30 drops in juice or water before meals, 3 times a day, 6 days a week.

Note:
Consult Health Practitioner if pregnant.




Gymnema sylvestre is one of the Asclepiad strains that grows in South-east Asia. Their therapeutic effects for treating diabetes mellitus, rheumatic arthritis and gout have been well known for a long time. However, the problem is that GS suppresses sweetness and tastes bitter. For this study, we chose Gymnema inodorum (GI) instead of GS, since it has an advantage that it does not suppress sweetness nor is it bitter in taste. Our studies suggest that the component of GI inhibits the increase in the blood glucose level by interfering with the intestinal glucose absorption process.

The effectiveness of GS4, an extract from the leaves of Gymnema sylvestre, in controlling hyperglycemia was investigated in 22 Type 2 diabetic patients on conventional oral anti-hyperglycemic agents. GS4 (400 mg/day) was administered for 18-20 months as a supplement to the conventional oral drugs. During GS4 supplementation, the patients showed a significant reduction in blood glucose, glycosylated haemoglobin and glycosylated plasma proteins, and conventional drug dosage could be decreased. Five of the 22 diabetic patients were able to discontinue their conventional drug and maintain their blood glucose homeostasis with GS4 alone. These data suggest that the beta cells may be regenerated/repaired in Type 2 diabetic patients on GS4 supplementation. This is supported by the appearance of raised insulin levels in the serum of patients after GS4 supplementation.

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